[Protection of the human skin and eyes against solar UV-radiation]. / Zashchita kozhi i glaz cheloveka ot UF-izlucheniia solntsa.

Methods of skin and eye protection from solar UV-radiation are described. Data concerning UV-ray absorption by different types of clothing as well as various ointments, including new agents, are presented. When considering possibilities of eye protection from UV-radiation, characteristics of different filters are discussed, Soviet-made sun-glasses are listed, and eye protection drugs are indicated. In this context, approaches to eye protection of pilots and cosmonauts are outlined.

[Damage to the skin and eyes from solar UV-radiation]. / Povrezhdeniia kozhi i glaz UF-izlucheniem solntsa.

Skin and eye injuries induced by solar UV-radiation are discussed. Penetration of this radiation into the skin, mechanisms of skin erythema and pigmentation as well as skin burns are described. The spectral sensitivity of the skin to UV-rays of different wavelengths as well as threshold doses are presented. Emergence of UV-radiation induced eye injuries, their clinical picture and threshold doses of deleterious effects which produce injuries of the eye conjunctiva, cornea, and core are described.

[Changes in cytospectrophotometric characteristics and configuration of erythrocytes exposed to constant electric current]. / Izmeneniia tsitospektrofotometricheskikh kharakteristik i konfiguratsii éritrotsitov pri vozdeistvii postoiannogo élektricheskogo toka.

The action of constant current (0.01-3 A, 1-25 V) on human blood during varying exposures induced intensified plaque-formation in the preparations of the local hemolysis test, and transformation of red blood cells-discocytes into echino- and poikilocytes that was attended by pronounced changes in the extent, intensity of light diffusion, natural green and red luminescence of red blood cells caused by flavoproteins and metalloporphyrins. The growth of the last factor has evidenced the presence of significant destructive changes in the cells as a result of constant current direct action on red blood cells.

[Role of photoreactivation in case of corneal damage from ultraviolet radiation]. / Rol' fotoreaktivatsii v vozniknovenii povrezhdenii rogovoi obolochki ul'trafioletovym izlucheniem.

Experimentally, the role of photoreactivation in appearance of ultraviolet lesions of corneal epithelium has been studied in white rats. The lesions were induced by ultraviolet radiation, 0.28 mcm wavelength, and intensive (20000 lx) light of a xenon lamp with light filters was used as a reactivating light. The damaging dose as a reactivating light. The damaging dose of ultraviolet radiation by photokeratitis criterion was 11 +/- 2.4 mJ.cm-2. Simultaneous illumination of the animal eye by the damaging ultraviolet light and reactivating radiation exceeded a damaging threshold to 18 +/- 4.0 mJ.cm-2. The results obtained have convincingly shown, that photoreactivation, as a phenomenon, manifested itself in the corneal epithelium and was detected by biomicroscopy of the eye with epithelium staining by a 1% fluorescein solution.

[Transformation and ultrastructural changes in erythrocytes in local hemolytic reactions]. / Transformatsiia i ul'trastrukturnye izmeneniia éritrotsitov v reaktsiiakh lokal'nogo gemoliza.

Human and animal cell transformation in the local autoimmune hemolysis and stimulated erythrodieresis is enhanced during plaque formation, which fact is parallelled by a growing number of local hemolysis sites and increase of these sites' area in the preparations. Echino-poikilospherocytic transformation and ultrastructural changes in the red cells in these tests are explained by enhanced synthesis of autoantibodies-hemolysins by autoplaque-forming cells, as well as by the osmotic processes and destabilization of the cellular membrane system, including the leukocytic membranes.

[The quantitative erythrogram and the possibiilty of its clinical and experimental use]. / Kvantitativnaia éritrogramma i vozmozhnost' ee ispol'zovaniia v klinike i éksperimente.

Quantitative erythrogram, i. e. the per cent distribution of the cells of different shapes into 5 groups (discocytes, stomatocytes, echinocytes, poikilo- and schizocytes, hemolytic forms) is recommended to be taken into consideration when estimating the red cell transformation in clinical and experimental studies. The classifying morphologic signs of about forty erythrocyte forms are described, and examples of the quantitative erythrogram use are given.

[Regional redistribution of the blood after 7 and 30 days of hypokinesia in rats]. / Regionarnye pereraspredeleniia krovi posle 7- i 30-sutochnoi gipokinezii u krys.

An exposure of rats to hypokinesia was accompanied by a significant increase in the total amount of circulating blood per unit body mass. The percentage blood content was increased in the myocardium, lungs, liver, kidneys, small intestine, muscles and bones of the chest. The percentage blood content was decreased in the skin of different body segments, muscles and bones (except of the chest), tail, viscera (large intestine, adrenals, spleen, bladder, testes). The major regional redistribution of blood was its displacement into the liver.

[Effect of long-term hypokinesia on the course of acute aseptic inflammation]. / Vliianie dlitel'noi gipokinezii na techenie ostrogo asepticheskogo vospaleniia.

White rats were kept under hypokinetic conditions for 15, 28 and 60 days. By physiological, microbiological and morphological methods it was demonstrated that hypokinesia had an adverse effect on the clinical development of aseptic inflammation. This effect included a more marked edema and its delayed resorption, an increased temperature of the inflammatory focus, an increased number of infectious complications, involving necrotic foci and ulcerations, enhanced alteration processes, inhibited formation of the demarcation zone, generalized inflammation, etc. Resistance to phlogogenic stimulation fell down to a minimum during the first two hypokinetic weeks; afterwards it gradually returned to normal but did not recover entirely even after 60 days of hypokinesia. Following 15- and 28-day hypokinetic experiments resistance to phlogogenic agents reached the pretest level within 7 days of the recovery period.