[Species specificity of the isoenzyme profile of lactate dehydrogenase in organs of rodents of various ecogenesis]. / Vidovaia spetsifichnost' izofermentnykh profilei laktatdegidrogenazy organov gryzunov razlichnogo ékogeneza.

Separation of isoenzymes of lactate dehydrogenase (LDH, EC. 1.1.1.27) in extracts of heart, kidney, liver, spleen, lungs of nutrias, chinchillas by agar gel electrophoresis reveals a species specificity in ratio of electrophoretic fractions of the enzyme. The isoenzymes of LDH were seem to play an important role in adaptation of fur animals to environmental conditions. It has been shown that in semiaquatic mammals--nutrias, the relative content of the A-subunits in the isoenzymatic spectrum of LDH in organs was increased as compared with terrestrial animals--chinchillas, whereas relative content of B-subunits in these organs of chinchillas was very high. This is an example of subtle biochemical specialisation of function at molecular level to environmental conditions.

[Isozyme patterns of lactate dehydrogenase from tissues of mink and arctic fox during postnatal development]. / Izofermentnye spektry laktatdegidrogenazy organov norok i pestsov v postnatal'nom razvitii.

Isozymes of lactate dehydrogenase extracted from heart, kidney, and liver of mink (Mustela vison Briss.) and Arctic fox (Alopex lagopus L.) during postnatal development were separated by agarose gel electrophoresis. Tissue-specific isozyme pattern of lactate dehydrogenase can be revealed at the age of one month, while the definitive pattern is formed at the age of four months. The isozyme patterns of lactate dehydrogenase in the studied tissues of mink and Arctic fox share the properties specific for animal species of various ecogenesis.

[Effect of mutilus hydrolysate in Aleutian disease of minks]. / Effekt gidrolizata iz midii pre aleutskoi bolezni norok.

Addition of mutilus hydrolysate MIGI-K to rations of minks with virus plasmacytosis involving grave immunological disorders led to changes in the ratio of serum protein fractions and in the differential blood count. Normalization of the protein spectrum of minks with Aleutian disease apparently inhibits the development of disorders caused by hypergammaglobullnemia.