The Photoluminescence Properties of Dendritic Phosphors Ca2Gd8(SiO4)6O2:A (A = Eu³âº and Dy³âº) for White-Light-Emitting Diodes.

Ca2Gd8(SiO4)6O2:A (A = Eu³âº and Dy³âº) phosphors were successfully synthesized by the sol-gel process. X-ray diffraction (XRD), thermogravimetric and differential thermal analysis (TG-DTA), Fourier transform infrared spectroscopy (FT-IR), transmission electron microscopy (TEM), and photoluminescence spectra were used to characterize the resulting phosphors. The results of XRD indicated that the diffraction peaks of the phosphor powder agreed well with the standard card. Under short ultraviolet excitation, the doped rare earth ions (A) showed their characteristic emis- sions in Ca⁺Gd8(SiO4)6O2 phophors, i.e., 5D0 --> 7F2 (615 nm) for Eu³âº and 4F9/22 --> 6H15/2 (483 nm) and 4F9/2 --> 6H13/2 (575 nm) for Dy³âº. The optimum concentration for doped rare earth ions was determined to be 9 mol% Eu³âº and 3 mol% Dy³âº of Gd³âº in Ca2Gd8(SiO4)6O2

The Photoluminescence Properties of CaYAI3O7:Ce³âº, Tb³âº, Sm³âº Prepared by the Sol-Gel Process.

This paper reports the synthesis and luminescence properties of rare earth doped CaYAI3O7 prepared via a sol-gel process. The phosphors have intense luminescence. In Ce³âº/Tb³âº co-doped materials, there is energy transfer from Ce³âº to Tb³âº that enhances the luminescence of Tb³âº. These materials may find applications in solid state lighting and radiation detection.

The toxic reactions of all-trans retinoic acid occuring in the treatment of acute promyelocytic leukemia / 中国癌症杂志

Purpose:To observe the toxic reactions of all trans retinoic acid in the treatment of acute promyelocytic leukemia. Methods:Analysis of toxic reaction was done for 38 patients who suffered from acute promyelocytic leukemia treated with all trans retinoic acid. Results:In the treatment of acute promyelocytic leukemia with all trans retinoic acid, the common toxic reactios are dry mouth, skin desiccation, desquamation, rhagadia (100%). 19 patients had gastro intestinal symptoms with higher ALT(50%),18 patients had hyperleukocytosis syndrome (47.5%), 16 patients had headache and dizziness (42.4%),8 patients had pain in muscle and joint (21.2%). The rare toxic reactions included 4/38 who had intracranial hypertension syndrome, 2/38 had hyperglycemia, 1/38 and hyperhistaminemia syndrome. Conclusions:The toxic reactions of all trans retinoic acid in the treatment of acute promyelocytic leukemia usually have dry mouth ,skin desiccation, desquamation, rhagadia, gastro intestine symptom with higher ALT, and hyperleukocytosis syndrome ets. THe treatment with lower dose of all trans retinoic acid(20～30 mg/d)for acute promyelocytic leukemia could decrease the toxic reactions without influencing its effectiveness.