[Ocular hydrodynamics after phacoemulsification cataract surgery in patients with primary open-angle glaucoma]. / Osobennosti gidrodinamiki posle fakoemul'sifikatsii u patsientov s pervichnoi otkrytougol'noi glaukomoi.

AIM: To investigate parameters of intraocular fluid outflow after phacoemulsification of cataract in patients with primary open-angle glaucoma (POAG). MATERIAL AND METHODS: The study involved 153 cataract patients (153 eyes), most of whom also had POAG. The patients were divided into three groups: group 1 - 51 patients (51 eyes) with cataract but no glaucoma, group 2 - 51 patients (51 eyes) with cataract and early POAG, and group 3 - 51 patients (51 eyes) with cataract and advanced POAG. All of them underwent non-contact pneumatic tonometry, tonography, and tonography with vacuum compression (Glau-Test60) before and after surgery (phacoemulsification of cataract with implantation of a posterior chamber intraocular lens (IOL)). The follow-up period was 6 months. RESULTS AND DISCUSSION: Phacoemulsification surgery with IOL implantation was able to reduce intraocular pressure in non-glaucoma cataract patients by 3 to 5 mmHg in 21.57% of cases and by more than 5 mmHg in 58.8% of cases. In patients with early and advanced glaucoma, a 3 to 5 mmHg decrease occurred in 33.3% and 13.7% of cases, respectively, and a more than 5 mmHg decrease - in 49.02% and 56.9%, respectively. This reduction is due to activation of intraocular fluid outflow. CONCLUSION: A postoperative increase in trabecular and uveoscleral outflow has been detected in all groups. The change was more significant in non-glaucoma patients and those with early disease.

[Uveitis, caused by Echo-11 virus]. / Uveit, vyzvannyi virusom Echo-11.

Analysis of clinical manifestations of a total enteroviral infection with ECHO-11 virus-induced uveitis in 58 infants aged 2 weeks to 1.5 years has shown that uveitis develops on days 1-10 of the disease and is associated with a weak injection, endothelial edema, hyperemia and edema of the iris rapidly eventuating in destruction of the pigment lamina and fenestration. Posterior synechiae, pupil deformations, grave uveitis with hypotonia of 4-10 mm Hg are rapidly developing. A two-year follow-up has shown marked hemo- and hydrodynamic disorders, retarded growth of the involved eye, development of grave complications in 60 percent of patients. The most characteristic late complications were iridal fenestration, pseudocoloboma-type pupil deformation, formation of prelental films, often pigmented. The condition is associated with active production of virus-neutralizing antibodies in titers of 1:4 to 1:16384. No relationship between the clinical course and antibody titers was revealed.