RESUMO
An overview of the metabolic response to acute traumatic brain injury is presented. The consequences of hypermetabolism, hypercatabolism, and an altered immune function are discussed. Once a person with acute traumatic brain injury develops this hyperdynamic state, the resultant excessive protein breakdown ensues. This can lead to malnutrition. The feeding methods used to prevent malnutrition are discussed, along with the proper alimentation to provide to diminish the hyperdynamic state and improve immune function.
Assuntos
Lesões Encefálicas/metabolismo , Lesões Encefálicas/terapia , Apoio Nutricional/métodos , Doença Aguda , Fatores Etários , Algoritmos , Estatura , Peso Corporal , Lesões Encefálicas/complicações , Lesões Encefálicas/imunologia , Árvores de Decisões , Metabolismo Energético , Feminino , Escala de Coma de Glasgow , Humanos , Masculino , Avaliação Nutricional , Distúrbios Nutricionais/etiologia , Distúrbios Nutricionais/prevenção & controle , Necessidades Nutricionais , Seleção de Pacientes , Fatores SexuaisRESUMO
Hypopituitarism secondary to penetrating head trauma is extremely rare, and its diagnosis may be delayed for several years. We present a patient who developed hypopituitarism secondary to a transfacial gunshot wound and who experienced damage to the hypophysis secondary to transmission of energy and bullet fragments. The importance of a computerized tomographic scan of the head in facial gunshot wound is discussed.
