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1.
Lik Sprava ; (7-8): 59-62, 1999.
Artigo em Ucraniano | MEDLINE | ID: mdl-10672691

RESUMO

Forty-one couple with the communicative form of sexual dysadaptation including sexual aversion in which men suffered from different forms of neurosis (neurasthenia, hysteric neurosis, obsessional neurosis) were examined using the method of systems-structural analysis. The results of the studies made revealed causes and mechanisms of development of the considered form of sexual dysadaptation, with manifestations and consequences of the above dysadaptation-related disorders of sexual health of the family described in some detail. A relation was found out between the form of neurosis in men and development of sexual dysadaptation. The necessity is substantiated to consider the revealed regularities in making a diagnosis or performing a correction of aversion in male subjects with neurosis.


Assuntos
Adaptação Psicológica , Comunicação , Transtornos Neuróticos/complicações , Disfunções Sexuais Psicogênicas/etiologia , Adulto , Doença Crônica , Conflito Psicológico , Humanos , Masculino , Pessoa de Meia-Idade , Transtornos Neuróticos/diagnóstico , Transtornos Neuróticos/psicologia , Disfunções Sexuais Psicogênicas/diagnóstico , Disfunções Sexuais Psicogênicas/psicologia
2.
Biull Eksp Biol Med ; 103(6): 650-3, 1987 Jun.
Artigo em Russo | MEDLINE | ID: mdl-3593943

RESUMO

The experiments on rats have shown that bilateral administration of kainic acid (0.1-0.15 microgram) into the rostral parts of caudate nuclei led to the development of hypokinesia and rigidity. An increase in the electrical activity--the formation of the generator of pathologically increased excitement (GPIE) was noted in a zone of kainic acid injection. Rigidity and hypokinesia were attenuated and the GPIE activity was depressed after cyclodol (1-10 mg/kg) or L-DOPA (100-200 mg/kg) administration. Combined administration of cyclodol (2 mg/kg) and L-DOPA (50 mg/kg) induced potentiated antiparkinsonian effect. Dopamine microinjections into the GPIE area depressed its activity and abolished rigidity and hypokinesia. These data suggest that the Parkinson syndrome develops under the influence of GPIE induced by kainic acid administration into caudate nuclei.


Assuntos
Modelos Animais de Doenças , Ácido Caínico/farmacologia , Doença de Parkinson Secundária/induzido quimicamente , Animais , Núcleo Caudado , Avaliação Pré-Clínica de Medicamentos , Eletrodos Implantados , Eletroencefalografia , Feminino , Injeções , Levodopa/uso terapêutico , Masculino , Doença de Parkinson Secundária/tratamento farmacológico , Ratos , Fatores de Tempo , Triexifenidil/uso terapêutico
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