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2.
Chirurgie ; 120(6-7): 385-7, 1994.
Artigo em Francês | MEDLINE | ID: mdl-7768131

RESUMO

Among entero-vascular fistulae, the duodenocaval type is exceptional. A case arising in a patient operated 10 months ago from a right kidney cancer, is reported. The clinical feature associated a septicemia to an intestinal hemorrhage. The diagnosis of entero-vascular fistula was performed at the operating time. In the world literature only 8 cases of duodeno-caval fistulae have been previously reported. The injury of duodenocaval area is the most frequent etiology of the communication causing moderate digestive hemorrhage and understood septicemia. The imaging explorations may suspect the diagnosis that is confirmed baparotomy. The treatment is always a surgical one: closure of digestive fistula by intestinal patch and suture of vascular defect. The prognosis depends on experience and skill of the surgeon and remains serious.


Assuntos
Duodenopatias/diagnóstico , Fístula Intestinal/diagnóstico , Veia Cava Inferior , Idoso , Duodenopatias/cirurgia , Fístula/diagnóstico , Fístula/cirurgia , Humanos , Fístula Intestinal/cirurgia , Masculino , Doenças Vasculares/diagnóstico , Doenças Vasculares/cirurgia
3.
J Chir (Paris) ; 115(8-9): 465-72, 1978.
Artigo em Francês | MEDLINE | ID: mdl-721918

RESUMO

The authors relate 15 cases of blood coagulation disturbances among three groups of animals after 80%, 90% or total hepatectomy. Animals in group I, after 80% hepatectomy, were sacrified between the 10th and 30th postoperative days. Group II animals, with 90% hepatectomy, survived for periods ranging from 18 to 24 hours. In group III, after total hepatectomy, survival varied between 30 and 40 hours. The coagulation disturbances which were observed following 80% hepatectomy were spontaneously compatible with the survival of the animals. Following 90% and total hepatectomy the resulting disturbances were constantly severe enough to limit survival time. Supportive procoagulant therapy associated with sequential heparin administration seems justified by the insufficient biosynthesis of clotting factors and their unavoidable consumption as a result of localized then disseminated intravascular coagulation and secondary fibrinolysis.


Assuntos
Transtornos da Coagulação Sanguínea/etiologia , Hepatectomia/efeitos adversos , Animais , Transtornos da Coagulação Sanguínea/fisiopatologia , Fatores de Coagulação Sanguínea/biossíntese , Coagulação Intravascular Disseminada/etiologia , Cães , Hemostasia
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