The Implications of Stress Among Correctional Officers: A Summary of the Risks and Promising Intervention Strategies.

This paper provides a detailed summary and discussion of the concept of stress and how it relates to the health, wellbeing, and performance of corrections officers. In line with these objectives, we focus on three areas: (1) providing a more detailed definition of the concept of stress; (2) a discussion of the ways that increased exposure to stress may impact corrections officers' physical and mental health; and (3) a summary of prevention and intervention strategies that are relevant for corrections officers and have shown promise in dampening the consequences of increased stress exposure. More in-depth knowledge of the concept of stress and the underlying processes that link stress to negative outcomes will provide policy makers and corrections departments with an understanding of the characteristics of prevention and intervention strategies that are expected to be most effective in limiting the consequences of stress.

Adrenocortical Responses to Daily Stressors Are Calibrated by Early Life Adversity: An Investigation of the Adaptive Calibration Model.

Studies examining the impact of early adversity on physiological responsivity to environmental challenges in later life yield a complex pattern of findings and ambiguity regarding the direction of effect, with some studies reporting heightened responses and others reporting dampened responses. One potential reason for these mixed findings is an oversimplified theoretical model surrounding the connection between early life stressor exposure and subsequent stress responsivity. The adaptive calibration model offersa contemporary set of assumptions aimed at providing a better understanding of the ways that early life experiences shape the stress response system to better align with current and future environments. The current study utilized a large subsample from the National Study of Daily Experiences (N = 1,605) to examine the extent to which the association between daily stressor exposure and cortisol levels varies across levels of early life adversity. Results revealed that those individuals who experienced extremely low levels of early life adversity displayed the greatest increase in cortisol levels across the day as daily stressor exposure increased. Alternatively, those individuals who experienced extremely high levels of early life adversity displayed almost no change in diurnal production of cortisol as daily stressor exposure increased. The results are discussed within the evolutionary-developmental context of the adaptive calibration model along with suggestions for future research.

Racial and ethnic differences in the availability of community medical services after leaving prison.

INTRODUCTION: Incarcerated individuals experience increased health problems, presenting additional challenges as they leave prison and re-enter the community. These challenges are disproportionally experienced by racial and ethnic minorities. Despite these trends, little is known regarding the availability of medical services within the communities to which incarcerated individuals return. METHODS: We examined all prison returns in the state of Florida between 2008 and 2017. We examined the odds of returning from prison to a community that is formally designated as medically underserved by the Health Resources and Services Administration. We also examined whether Florida communities with a greater proportion of racial and ethnic minority returns were more likely to be designated as medically underserved. RESULTS: Overall, each SD increase in community return rate resulted in a 20% increase in the odds of medical underservice designation. For each SD increase in the proportion of black and Latino returns, the odds of medical underservice designation increased by 50% and 14%, respectively, compared with the proportion of white returns. DISCUSSION: Within Florida, previously incarcerated individuals are more likely to return to communities with limited availability of medical services. These findings are even more pronounced for communities with more black returnees. Previously incarcerated individuals are more likely to return to communities that lack the medical infrastructure required to address their unique healthcare needs, potentially leading to worsened health, and increased racial and ethnic health disparities.

Adolescent Violent Delinquency Associated With Increased Emergency Department Usage in Young Adulthood.

Limited research has examined the extent to which adolescent delinquency predicts healthcare usage in young adulthood, including emergency department (ED) visits. This study used data from 3,310 adolescents (52.05% female; mean age at Wave I = 16.04 years) from the sibling subsample of the National Longitudinal Study of Adolescent to Adult Health (Add Health). We examined whether adolescent delinquency at Wave I predicted ED visits at Wave III using sibling fixed effects models to adjust estimates for within-family unobserved heterogeneity. Increased violent, but not nonviolent, delinquency predicted a higher number of ED visits in early adulthood in the sibling fixed effects models. To our knowledge, this is the first study to examine the relationship between delinquency and ED usage using a sibling fixed effects design. Findings demonstrate that violent adolescent delinquency may increase healthcare usage and suggest the potential role of healthcare providers in improving outcomes for delinquent youth.

Social Support as a Protective Factor for Alcohol Use Disorders: Results from a Nationally Representative Family History Study.

AIMS: The current study examined the buffering effect of social support on the relationship between family history and alcohol use disorder symptoms (AUDsx). METHODS: The current study analyzes data from Waves 1 and 2 of the National Epidemiologic Survey of Alcohol and Related Conditions (N = 34,653). Count of AUDsx were measured using the Alcohol Use Disorder and Associated Disabilities Interview Schedule-DSM-IV Version, the independent variable was a weighted density measure of family history of AUDsx and the moderating variable was social support measured using the 12-item Interpersonal Support Evaluation List (ISEL-12). Negative binomial regression models were used to estimate the association between (1) family history and AUDsx and (2) social support and AUDsx. Average marginal effects were estimated to explore the buffering effect of social support on the association between family history and AUDsx. RESULTS: Family history was positively associated with AUDsx (incidence rate ratio (IRR) = 1.46, 95% CI = 1.39-1.54) and social support was negatively associated with AUDsx (IRR = 0.80, 95% CI = 0.74-0.86). The marginal effects of family history decreased at higher levels of social support, indicating a buffering influence of social support on the association between family history and AUDsx. CONCLUSIONS: Results reveal a buffering effect of social support, where greater levels of social support reduce the association between family history and AUDsx. These results indicate that the social context, and social support specifically, may be important for diminishing the risk of AUDsx.

Are Most Published Criminological Research Findings Wrong? Taking Stock of Criminological Research Using a Bayesian Simulation Approach.

This study uses Bayesian simulations to estimate the probability that published criminological research findings are wrong. Toward this end, we employ two equations originally popularized in John P.A. Ioannidis' (in)famous article, "Why Most Published Research Findings are False." Values for relevant parameters were determined using recent estimates for the field's average level of statistical power, level of research bias, level of factionalization, and quality of theory. According to our simulations, there is a very high probability that most published criminological research findings are false-positives, and therefore wrong. Further, we demonstrate that the primary factor contributing to this problem is the poor quality of theory. Stated differently, even when the overall level of research bias is extremely low and overall statistical power is extremely high, we find that poor theory still results in a high rate of false positives. We conclude with suggestions for improving the validity of criminological research claims.

Within-Individual Changes in Impulsivity and Sensation Seeking from Childhood to Early Adulthood and Educational Attainment.

Developmental perspectives recognize the importance of the development of impulsivity and sensation seeking for later life educational attainment. The current study examines the effect of developmental trajectories of impulsivity and sensation seeking across adolescence and into young adulthood on educational attainment in adulthood. The study sample consists of N = 5529 youth from a population-based sample from the United States (48.96% female; 46.95% Black or Hispanic). Latent growth curve models are used to examine associations between within-individual changes in impulsivity and sensation seeking and educational attainment. The findings point to the existence of multiple forms of risk taking during different developmental stages, with some offering a positive long-term effect on overall educational attainment.

An Examination of the Components of Toxic Stress in Childhood and Biological Markers of Physical Health in Emerging Adulthood.

Experiencing severe and enduring adversity in childhood without the support of adult figures has been linked to an extensive list of physical health outcomes. This finding is closely tied to the concept of toxic stress, which is regularly studied using a combination of sources, including childhood adversity, unmet basic needs, and unmet social needs. Despite these findings, previous work has typically compiled various sources associated with toxic stress into a single construct, limiting existing knowledge on the contribution of each individual source to physical health. To address these concerns, the current study utilizes data from the National Longitudinal Study of Adolescent to Adult Health to examine the association between independent and collective sources of toxic stress in childhood and individual differences in biomarkers tapping cardiometabolic functioning in emerging adulthood. Results indicate a significant association between a composite measure of sources of toxic stress and cardiometabolic risk, with subsequent models examining the independent influence of each source revealing that this association was largely driven by childhood adversity and unmet basic needs, but not unmet social needs. These findings suggest that the individual sources of toxic stress may differentially contribute to physical health outcomes.

The influence of adverse childhood experiences on internalizing and externalizing problems in early adulthood: Evidence of a gene × environment × sex interaction.

BACKGROUND: Although adverse childhood experiences (ACEs) have negative effects on subsequent wellbeing, questions remain regarding how and why they do so. Sex, environmental effects, and genetic influences may play a role in both one's exposure to ACEs as well as one's reactions to ACEs. OBJECTIVE: To understand the combined genetic and environmental influences on males' and females' exposure and reactions to ACEs, and to determine whether sex differences in offending and depressive symptoms were partially impacted by genetic influences. METHODS: We employed a sample of monozygotic twins (n = 217 pairs), same-sex dizygotic twins (n = 185 pairs), and same-sex full siblings (n = 446 pairs) from the National Longitudinal Study of Adolescent to Adult Health (N = 848 pairs) and estimated a series of multivariable biometric models. Participants were aged between 12 and 21 during Wave I, between 13 and 22 at Wave II, between 18 and 26 at Wave III, and 24 and 32 at Wave IV. RESULTS: First, there appears to be a stronger genetic influence on ACEs exposure among males than females. Second, genetic influences were stronger on offending among males and depression among females. Third, ACEs moderate the genetic influences on offending and depressive symptomology among males and females: among males, genetic influences on offending decreased as exposure to ACEs increased, while among females, genetic influences on depressive symptoms decreased as exposure to ACEs increased. CONCLUSIONS: Systematic sex differences in the exposure and reactions to ACEs are at least partially due to genetic differences. Exposure to ACEs is partially influenced by genetics among males, but not females, and the more male and females' experience ACEs, the less influence genes have on their offending and depressive symptomology, respectively.

Examining adverse childhood experiences among Native American persons in a nationally representative sample: Differences among racial/ethnic groups and race/ethnicity-sex dyads.

BACKGROUND: Existing research using nationally representative samples has provided valuable information regarding the prevalence and context of childhood adversity, but Native American persons have largely been absent from these studies. OBJECTIVE: We examined adverse childhood experiences (ACEs) among persons identifying as White, Black, Asian, Hispanic, and Native American in the NESARC, a longitudinal study (Wave 1: 2001-2002; Wave 2: 2004-2005) using a nationally representative sample from the United States. METHODS: Means tests and negative binomial regression were used to examine the prevalence and variety of ACEs across racial/ethnic groups and race/ethnicity-sex dyads. RESULTS: Native American persons reported the greatest average number and variety of ACEs than persons from any other racial/ethnic group, and reported the highest rates of physical abuse, sexual abuse, parental substance abuse, and witnessing violence than members of any other racial/ethnic category. Native American females reported the greatest rates of emotional abuse, while Native American males reported the greatest rates of physical neglect; the highest rates of parental substance use among the race/ethnicity-sex dyads were reported by both Native American females and males. Significantly higher rates of sexual violence were reported by Native American females compared to other groups; almost 1 in 4 Native American females reported sexual violence. CONCLUSIONS: Future research should make a concerted effort to broaden examinations of ACEs to include Native American respondents and to include measures of historical trauma and racial discrimination. Broader support for system change as well as increased development and use of culturally responsive prevention and intervention programming is likely necessary to reduce ACEs among Native American persons.

Neighborhood Exposure to Violence and Delinquency in the Kingdom of Saudi Arabia: A Partial Test of Social Structure and Social Learning Theory.

The social structure and social learning (SSSL) model for crime and deviance has received an impressive amount of empirical support in the United States and other Western industrialized countries. Comparatively, less research, however, has examined whether the SSSL model offers a viable framework for explaining variation in delinquent behavior in other geographic contexts, particularly, countries that place a stronger emphasis on social control stemming from both formal and religious sources. The current study addresses this void in the literature by examining a sample of youth from Saudi Arabia, a Middle Eastern country that enforces Sharia (a set of laws based in Islamic tradition) and strict gender roles. The association between neighborhood exposure to violence and risk for violent and nonviolent delinquent behavior was examined using structural equation models. Subsequent models were aimed at more closely examining the mediating role of delinquent peer association between neighborhood exposure to violence and violent and nonviolent delinquent behavior. Results indicate that males exposed to neighborhood violence are more likely to engage in violent and nonviolent delinquent behavior, whereas females are more likely to engage in violent, but not nonviolent, delinquent behavior. In line with the SSSL model, delinquent peer association fully mediates the direct effect of neighborhood exposure to violence on delinquent behavior in both males and females. Findings from the current study suggest that the SSSL model may provide a useful framework for explaining individual differences in delinquent behavior in Saudi Arabia.

The Association Between Genetic Predisposition and Parental Socialization: An Examination of Gene-Environment Correlations Using an Adoption-Based Design.

An extensive body of research has examined the role that genetic influences play in the development of antisocial behavior. Even so, there remains much that is unknown regarding the intersections among antisocial behavior, environments, and genetic influences. The current study is designed to shed some light on this issue by examining whether gene-environment correlations are present in the lives of adopted adolescents. More specifically, this article seeks to contribute to scholarship efforts aimed at understanding whether biological parents' antisocial behavioral phenotypes-behaviors often attributed to an increased likelihood of receiving a genetic propensity for antisocial behaviors-predict variation in environments that are experienced by their adopted-away offspring. To do so, the biological parents of adoptees were assessed and used to identify ways in which children elicit certain responses from their adoptive parents based, in part, on their genotype. Correlational analyses were calculated on a sample of adoptees (the final analytic sample ranged between n = 229 and n = 293) drawn from the National Longitudinal Study of Adolescent to Adult Health (Add Health). The results of the study revealed very little evidence of gene-environment correlations. The implications of these findings are considered.

A longitudinal examination of the association between intelligence and rearrest using a latent trait-state-occasion modeling approach in a sample of previously adjudicated youth.

Recidivism remains a serious issue in the modern criminal justice system, with over 80% of those previously incarcerated being rearrested within 9 years of release (Alper, Durose, & Markman, 2018). Although previous studies have identified risk factors that increase the probability of rearrest, much remains unknown regarding the full constellation of risk factors. One potential risk factor that has received limited attention is intelligence, as individuals with lower IQ scores have been found to be more likely to come into initial contact with the criminal justice system. Collectively, previous studies have provided preliminary evidence of intelligence as a risk factor for rearrest but have not fully explored this association. More specifically, it remains unclear whether the association between IQ and recidivism persists after controlling for time-invariant, individual-specific sources of variance in criminal behavior. The current study aimed to address this limitation and more closely examine the longitudinal association between IQ and rearrest with data from the Pathways to Desistance Study (N = 1,331 individuals). To distinguish variance in intelligence from time-stable, individual-specific variance in criminality, we estimated a latent trait-state-occasion model. A subsequent series of survival models, which included the previously estimated measure of criminality as a covariate, revealed a small and negative association between IQ and rearrest (hazard ratio = .95; 95% confidence interval [.92; .98]), suggesting that IQ may play only a minor role in recidivism. (PsycINFO Database Record (c) 2019 APA, all rights reserved).

Co-twin relationship quality as a moderator of genetic and environmental factors on urinary cortisol levels among adult twins.

Previous research has indicated that genetic and environmental factors shape physiological activity. Cortisol levels, in particular, have received significant attention, with studies indicating substantive heritability estimates across various sampling techniques. A related line of research has indicated that genetic and environmental factors that explain variability in cortisol levels may vary across context and experiences by way of gene-environment interactions (G × Es). Despite these findings, a limited number of studies have examined the extent to which interpersonal relationships may operate as a moderator. The current study focused on co-twin relationship quality as a source of moderation, as twins are more likely to have contact with one another and to form close, interpersonal relationships with their co-twin relative to singleton siblings. Using a sample of 298 adult twins from the National Survey of Midlife Development in the United States (MIDUS), we examined the extent to which genetic and environmental factors that explain variability in urinary cortisol levels varied across levels of co-twin relationship quality. The heritability of cortisol levels was greater and nonshared environmental influences were lower at greater levels of relationship quality. These findings suggest that the heritability of cortisol may vary across context, and positive relationships with others may moderate such factors.

Adverse childhood experiences and deleterious outcomes in adulthood: A consideration of the simultaneous role of genetic and environmental influences in two independent samples from the United States.

BACKGROUND: Adverse childhood experiences (ACEs) are a potent risk factor. Despite these findings, studies have also recognized the importance of considering additional sources of genetic and environmental influence that cluster within families. OBJECTIVE: To properly control for latent sources of genetic and within-family environmental influences and isolate the association between ACEs and the following outcomes in adulthood: physical health, depressive symptoms, educational attainment, income attainment, alcohol problems, and antisocial behavior. PARTICIPANTS AND SETTING: Two independent samples of twins and siblings from the United States: the Midlife Development in the United States (MIDUS) study (N = 862) and the National Longitudinal Study of Adolescent to Adult Health (Add Health; N = 3112). METHODS: Sibling comparison models, which control for latent sources of genetic and within-family environmental influences, were estimated to examine whether differential exposure to ACEs was associated with the examined outcomes. RESULTS: Families that experienced more adversity also experienced more deleterious outcomes. However, siblings that experienced more adversity were no more likely to experience deleterious outcomes than their co-siblings. However, greater exposure to ACEs was associated with increases in depressive symptoms (Add Health). Additional models revealed that the similarity between siblings from the same family stemmed from latent sources of within-family environmental influences not captured by traditional ACEs measures. CONCLUSIONS: Considering genetic influences and additional latent sources of within-family influences is crucial in isolating the effects of ACEs. Currently employed ACEs measures may not adequately capture the full range of impactful sources of family-level environmental influence.

Brain injury and adverse outcomes: a contemporary review of the evidence.

Recently, research focusing on the implications of brain injuries for deleterious outcomes spanning a wide range of developmental domains has flourished. Findings from this literature suggest that brain injury is a potent source of risk for negative outcomes including neurodegenerative diseases, cognitive impairment, behavioral problems, and psychiatric diagnoses. Despite this evidence, few studies have examined the extent to which these findings represent a causal relationship. This review outlines the expansive literature in this developing area and provides a discussion of potential threats to internal validity. Finally, suggestions for future research are provided with a particular emphasis on leveraging existing findings to better understand the role of brain injury in the development of deleterious outcomes.

Socioeconomic disadvantage and psychological deficits: Pathways from early cumulative risk to late-adolescent criminal conviction.

Early exposure to multiple risk factors has been shown to predict criminal offending, but the mechanisms responsible for this association are poorly understood. Integrating social-environmental and dispositional theories of crime this research investigated the capacity of family socioeconomic disadvantage and individual psychological deficits to mediate the association between childhood cumulative risk and late adolescent criminal convictions. Male participants in the 1986 Northern Finland Birth Cohort Study (n = 3414) were followed from the prenatal period through age 19-20. The data were analyzed by estimating a structural equation model of the hypothesized pathways. The results found support for both processes of influence, and the model sustained a statistically significant direct effect of cumulative risk on crime. Socioeconomic disadvantage and psychological deficits contribute to criminal offending independently and with roughly equal magnitude. The results point to the utility of both environmental and psychological interventions to prevent criminality among children at risk.

Lower catecholamine activity is associated with greater levels of anger in adults.

Previous research has revealed a consistent association between heart rate at rest and during stress and behavioral problems, potentially implicating autonomic nervous system (ANS) functioning in the etiological development of antisocial behavior. A complementary line of research has focused on the potential independent and interactive role of the two subsystems that comprise the ANS, the parasympathetic nervous system (PNS) and the sympathetic nervous system (SNS), on behavioral problems. The current study aims to contribute to the existing literature by examining the influence of heart rate (HR) reactivity, high-frequency heart rate variability (HF-HRV) reactivity, and catecholamine activity on a comprehensive measure of anger in a large, nationally-representative sample of adults from the United States. Results from a series of structural equation models (SEMs) revealed that catecholamine activity was most consistently linked to anger, while associations involving HR and HF-HRV reactivity were nonsignificant. Additional analyses revealed that HF-HRV did not significantly moderate the association between catecholamine activity and anger. These findings highlight the importance of SNS activity in the development of more reactive forms of aggression such as anger.

Long-term physical health consequences of perceived inequality: Results from a twin comparison design.

RATIONALE: Previous research has identified long-term exposure to stress as a risk factor for negative mental and physical health outcomes. This pattern of findings suggests that environmental stimuli that evoke feelings of stress or strain may also result in physiological responses, which may accumulate over the life course and ultimately increase the overall risk of various physical health conditions. This physiological "wear and tear" resulting from sustained levels of stress or strain has been previously operationalized as allostatic load (AL), a comprehensive indicator of stress exposure. OBJECTIVE: The current study examines the association between one potential environmental stressor-perceived inequality-and AL with a research design aimed at addressing both observed and unobserved sources of confounding; it also employs a more comprehensive AL measure (comprised of 24 biomarkers tapping seven physiological systems) than previous studies. METHOD: The biomarker twin sample from the Midlife Development in the United States (MIDUS) study was used to estimate a series of twin comparison models, which include controls for latent sources of influence that cluster within families. The sibling comparison models also included additional controls for lifestyle choices, overall physical health, and demographics which may confound the examined associations. RESULTS: The results revealed significant associations between greater perceptions of inequality and greater overall levels of AL. The association persisted even after including controls for both observed and unobserved influences that may confound the examined associations but was limited to more recent measures of perceived inequality. Associations involving earlier measures of perceived inequality, along with a lifetime measure, failed to reach conventional levels of significance. CONCLUSION: Perceived inequality appears to be a robust predictor of AL and potentially contributes to subsequent physical health problems, particularly for more proximate forms of perceived inequality.

How Cognitive Genetic Factors Influence Fertility Outcomes: A Mediational SEM Analysis.

Utilizing a newly released cognitive Polygenic Score (PGS) from Wave IV of Add Health (n = 1,886), structural equation models (SEMs) examining the relationship between PGS and fertility (which is approximately 50% complete in the present sample), employing measures of verbal IQ and educational attainment as potential mediators, were estimated. The results of indirect pathway models revealed that verbal IQ mediates the positive relationship between PGS and educational attainment, and educational attainment in turn mediates the negative relationship between verbal IQ and a latent fertility measure. The direct path from PGS to fertility was non-significant. The model was robust to controlling for age, sex, and race; furthermore, the results of a multigroup SEM revealed no significant differences in the estimated path coeficients across sex. These results indicate that those predisposed towards higher verbal IQ by virtue of higher PGS values are also predisposed towards trading fertility against time spent in education, which contributes to those with higher PGS values producing fewer offspring at this stage in their life course.