RESUMO
Injury to the inferior alveolar and lingual nerves may occur during removal of third molar teeth. An understanding of the possible mechanisms leading to nerve injury should reduce this occurrence. Careful assessment and documentation should follow a nerve injury, and early specialist referral considered. Although spontaneous recovery of low-grade nerve injuries is common, microneurosurgical techniques offer hope for more serious injuries.
Assuntos
Traumatismos dos Nervos Cranianos/etiologia , Traumatismos do Nervo Lingual , Dente Serotino/cirurgia , Extração Dentária/efeitos adversos , Traumatismos do Nervo Trigêmeo , Traumatismos dos Nervos Cranianos/cirurgia , Humanos , Distúrbios Somatossensoriais/diagnóstico , Distúrbios Somatossensoriais/etiologia , Distúrbios Somatossensoriais/cirurgiaRESUMO
Gardner's syndrome, with characteristic dental abnormalities and osteomas involving the jaws, is described in an adolescent male. The sinister aspect of the syndrome relates to the malignant potential of intestinal polyps and therefore the need for early diagnosis and prophylactic colectomy. The dentist may be the first health professional to have an opportunity to make an early diagnosis and referral of a patient with Gardner's syndrome, and should therefore be aware of the features of the syndrome that affect the teeth and jaws.
Assuntos
Síndrome de Gardner/patologia , Anormalidades Dentárias/patologia , Doenças Dentárias/patologia , Adolescente , Humanos , MasculinoRESUMO
The purpose of this double-blind randomized study was to assess recovery of mental function following reversal of midazolam-induced sedation with the specific antagonist flumazenil (R015-1788) or placebo following conservative dental procedures. Recovery was assessed using choice reaction time and critical flicker fusion threshold, both objective tests of psychomotor function; linear analogue sedation scores and simple memory tests. Assessments were repeated up to 3 h after administration of flumazenil or placebo to discover whether recovery was sustained or whether resedation occurred due to the short duration of action of flumazenil. Flumazenil in doses from 0.5 to 1.0 mg rapidly reversed the sedative and amnesic effects of a mean dose of 8.2 mg of midazolam without apparent evidence of subsequent resedation. Since recovery of mental function in the control group had ordinarily occurred 45 min after administration of placebo, routine reversal of midazolam sedation with flumazenil cannot be justified. Nevertheless, in cases of undue sedation persisting after dental treatment, flumazenil may be used with minimal risk of resedation occurring.
