[The effect of metoclopramide on the acid and pepsin secretion of patients with chronic gastritis and peptic ulcer]. / Vliianie metoklopramida na kisloto- i pepsinovydelenie u bol'nykh khronicheskim gastritom i iazvennoi bolezni.

The authors studied the effect of single intakes of metoclopramide on the acid and pepsin secretion in patients with chronic gastritis and duodenal ulcer. Peroral intake of 20 mg of metoclopramide during histamine-stimulated gastric secretion resulted in a distinct reduction of H ions and an insignificant reduction of pepsin secretion. Cerucal-initiated inhibition of acidity is accompanied by reduction of potassium concentration.

[The cytoprotective effect of prostaglandin E2 in duodenal ulcers]. / Tsitoprotektivnoe deistvie prostaglandina E2 pri iazvennoi bolezni.

Intragastric administration of a synthetic analog of prostaglandin E2 to patients with duodenal ulcers results in depression of the gastric acid production and in reduction of potassium level, leads to an elevation of the sialic acid debit and a rise of sodium concentration, not essentially changing calcium, chlorides, and pepsin levels. A combined analysis of gastric acid production and measurements of sialic acids, sodium and potassium detect the imbalance of the mucoid and bicarbonate secretion in peptic ulcers, this helping predict the disease course.

[Status of the electrolyte balance of the gastric juice during stimulation of secretion by histamine]. / Sostoianie élektrolitnogo balansa zheludochnogo soka pri stimuliatsii sekretsii gistaminom.

The content of electrolytes (Na+, K+, Ca2+) in human basal secrete has been determined before and after injection of histamine in gastric juice and insoluble mucus in healthy people. Electrolytes in the mucous membrane of the stomach, gastric juice and insoluble mucus have been determined in dogs with stomach fistula after Basow. Species peculiarities have been marked in electrolyte content change during gastric secretion.