RESUMO
PURPOSE: To differentiate by localized bioimpedance (L-BIA) measurements 24 h after injury, between tendinous, myotendinous junction (MTJ), and myofascial junction (MFJ) injuries, previously diagnosed by MRI exam. To evaluate by L-BIA, the severity of MTJ injuries graded from 1 to 3, and to determine the relationship between days to return to play (RTP) and L-BIA measurements. METHODS: 3T MRI and tetra polar L-BIA was used to analyzed 37 muscle injuries 24 h after injury in 32 male professional football players, (23.5 ± 1.5 kg m-2; 1.8 ± 0.1 m; 20-30 year.) between the 2016-2017 and 2017-2018 seasons. Muscle injuries were classified by The British Athletics Muscle Injury Classification (BAMIC). Percentage difference of L-BIA parameters [resistance (R), reactance (Xc), and phase angle (PA)] of the injured side were calculated considering contralateral non-injured side as the reference value. RESULTS: According to BAMIC classification and by MRI exam, we found tendinous (n = 4), MTJ (n = 26), and MFJ (n = 7) muscle injuries. In addition, MTJ injuries were grouped according to the severity of injury in grade 1 (n = 11), grade 2 (n = 8), and grade 3 (n = 7). Significant decrease (P < 0.01) was found in the L-BIA parameters R, Xc, and PA, in both MTJ and MFJ as well as in the different grades of MTJ injuries. In particular, in Xc (P < 0.001), which is related to muscle cell disruption. Regarding days to RTP, there was statistical significance among the three different grades of MTJ injuries (P < 0.001), especially when grade 1 was compared to grade 3 and grade 2 compared to 3. CONCLUSION: L-BIA is a complementary method to imaging diagnostic techniques, such as MRI and US, to quantify MTJ and MFJ injuries. In addition, the increase in the severity of the MTJ injury resulted in higher changes of the Xc parameter and longer time to RTP.
RESUMO
Aunque muchos autores la consideran una osteonecrosis, la enfermedad de Müller-Weiss es una displasia del escafoides tarsiano producida en la infancia, pero que se mantiene asintomática hasta la edad adulta. La fisiopatología es debida a un retraso en la osificación, asociado a una compresión de la porción lateral del escafoides tarsiano por el astrágalo lateralizado y en varo. El compromiso suele ser bilateral y simétrico
Although many authors consider Müller- Weiss syndrome as a osteonecrosis, it is a navicular dysplasia occurring in childhood that remains asymptomatic until adulthood. It is caused by a delay in the ossification of the navicular associated with the compression of the lateral portion of the tarsal navicular by the talus that is shifting laterally and in varus. There is usually bilateral and symmetrical involvement
