RESUMO
In Diabetes Mellitus the loss of capacity to regulate immunity, the reduction of pulmonary functions and the pro-thrombotic state determine the severity of COVID-19.
Assuntos
Betacoronavirus , Infecções por Coronavirus/complicações , Infecções por Coronavirus/fisiopatologia , Complicações do Diabetes/fisiopatologia , Pneumonia Viral/complicações , Pneumonia Viral/fisiopatologia , COVID-19 , Infecções por Coronavirus/imunologia , Complicações do Diabetes/imunologia , Diabetes Mellitus/imunologia , Diabetes Mellitus/fisiopatologia , Coagulação Intravascular Disseminada/etiologia , Coagulação Intravascular Disseminada/imunologia , Coagulação Intravascular Disseminada/fisiopatologia , Humanos , Modelos Biológicos , Neuroimunomodulação , Pandemias , Pneumonia Viral/imunologia , Síndrome do Desconforto Respiratório/etiologia , Síndrome do Desconforto Respiratório/imunologia , Síndrome do Desconforto Respiratório/fisiopatologia , Fatores de Risco , SARS-CoV-2 , Trombose/etiologia , Trombose/imunologia , Trombose/fisiopatologiaRESUMO
We have investigated plasmonic excitations at the surface of Bi_{2}Se_{3}(0001) via high-resolution electron energy loss spectroscopy. For low parallel momentum transfer q_{â¥}, the loss spectrum shows a distinctive feature peaked at 104 meV. This mode varies weakly with q_{â¥}. The behavior of its intensity as a function of primary energy and scattering angle indicates that it is a surface plasmon. At larger momenta (q_{â¥}~0.04 Å^{-1}), an additional peak, attributed to the Dirac plasmon, becomes clearly defined in the loss spectrum. Momentum-resolved loss spectra provide evidence of the mutual interaction between the surface plasmon and the Dirac plasmon of Bi_{2}Se_{3}. The proposed theoretical model accounting for the coexistence of three-dimensional doping electrons and two-dimensional Dirac fermions accurately represents the experimental observations. The results reveal novel routes for engineering plasmonic devices based on topological insulators.
