RESUMO
A review is given of the literature concerning the physiological and pathophysiological changes in humans, caused by sustained acceleration. After definition of the acting forces and an introduction into terminology, circulatory and respiratory mechanisms are described which are active under sustained acceleration. The origin of visual disturbances associated with acceleration is discussed. Acceleration tolerance is influenced by magnitude, duration, direction, and rate of application of G-forces together with environmental conditions and the condition of the subject. Acceleration protection may be achieved by technical devices (anti-g suits), voluntary manoeuvres and change of posture. Recent work dealing with high sustained +Gz (HSG) is reviewed and the associated problems are discussed.
Assuntos
Aceleração/efeitos adversos , Circulação Sanguínea/fisiologia , Pressão Sanguínea/fisiologia , Hipergravidade/efeitos adversos , Fenômenos Fisiológicos Respiratórios , Volume Sanguíneo/fisiologia , Trajes Gravitacionais , Frequência Cardíaca/fisiologia , Humanos , Consumo de Oxigênio/fisiologia , Postura/fisiologia , Inconsciência/prevenção & controle , Manobra de ValsalvaAssuntos
Aceleração , Medicina Aeroespacial , Fenômenos Biomecânicos , Estatura , Peso Corporal , Centrifugação , Humanos , Masculino , Postura , VibraçãoRESUMO
A crystalline solid, formed by oxidation of phthalocyanatomanganese(II) in pyridine, has been identified as phthalocyanatopyridinemanganese(III)-micro-oxo-phthalocyanatopyridinemanganese(III)dipyridinate(C(74)H(42)Mn(2)N(18)O2C(5)H(5)N) byx-ray diffraction methods. This novel molecule consists of two manganese complexes joined by an essentially linear Mn-O-Mn bridge. Its structure may have some relation to oxidation processes in biological systems.
