Temporal trends in the spatial-scale contributions to black carbon in a Middle Eastern megacity.

The analysis of high-resolution changes in black carbon (BC) concentrations was examined to distinguish and quantify various spatial-scale contributions to BC concentrations from nearby sources within 1 km distance to ranges of emission sources distributed over a larger city scale spanning tens of kilometers. Our analysis illustrated that BC emissions on the neighborhood scale only contribute a minor fraction (~15%) to total BC concentrations in the megacity of Tehran. Approximately 62% of the total black carbon is part of the city emissions, and around 23% is transported into the city from local nearby surroundings. Our analysis in highly polluted areas, including industrial and traffic hotspots in Tehran, demonstrated that the contributions of the urban mixture were relatively smaller than the average (~56%) in highly polluted areas; however, larger local-scale (~30%) contributions were observed in these areas. Our analysis in traffic hotspot areas also demonstrated significantly smaller contributions of BC from neighborhood surroundings (~9%). These results imply that the city-scale BC emissions in Tehran are a major contributor to BC exposures even in locations with local high-emitting sources. Polar annulus analysis of BC from city-scales in Tehran showed a mixture of hotspot locations ranging from north to easterly directions implying that city-scale emissions contribute to wider pollution plume expansions and larger-scale transport and vertical mixing corresponding to mixtures of emitters located further away.

Investigations into factors affecting personal exposure to particles in urban microenvironments using low-cost sensors.

Epidemiological studies have linked outdoor PM2.5 concentrations to a range of health effects, although people spend most of the time indoors. To better understand how individuals' exposure vary as they move between different indoor and outdoor microenvironments, our study investigated personal PM2.5 exposure and exposure intensity of 14 adult volunteers over one week (five weekdays and one weekend), using low-cost personal monitors, recording PM2.5 concentrations in 5 min intervals. Further, the study evaluated community perception of air pollution exposure during the recruitment and engagement with the volunteers. We found that people with tertiary education across all ages had greater interest in participating, with younger people being interested regardless of the level of education. The derived exposures and exposure intensities differed between weekdays and the weekend due to larger variations in individuals' daily routines. In general, time spent at home and engaged in indoor activities was associated with the highest personal PM2.5 exposure and exposure intensity on both, week and weekend days, implying the significance of both duration of the exposure and the indoor PM2.5 concentrations. The results showed no relationship between personal exposures and indoor characteristics of home (ventilation, building age and cooktop), which are expected to be due to the study's small sample size. The observed PM2.5 > 10 µg m-3 were significantly higher for distances <50 m to the roads for both major and minor roads, and were observed in areas with <16% open space, which were also close to a major road.

Effects of exposure to ambient ultrafine particles on respiratory health and systemic inflammation in children.

It is known that ultrafine particles (UFP, particles smaller than 0.1 µm) can penetrate deep into the lungs and potentially have adverse health effects. However, epidemiological data on the health effects of UFP is limited. Therefore, our objective was to test the hypothesis that exposure to UFPs is associated with respiratory health status and systemic inflammation among children aged 8 to 11 years. We conducted a cross-sectional study among 655 children (43.3% male) attending 25 primary (elementary) schools in the Brisbane Metropolitan Area, Australia. Ultrafine particle number concentration (PNC) was measured at each school and modelled at homes using Land Use Regression to derive exposure estimates. Health outcomes were respiratory symptoms and diagnoses, measured by parent-completed questionnaire, spirometric lung function, exhaled nitric oxide (FeNO), and serum C reactive protein (CRP). Exposure-response models, adjusted for potential personal and environmental confounders measured at the individual, home and school level, were fitted using Bayesian methods. PNC was not independently associated with respiratory symptoms, asthma diagnosis or spirometric lung function. However, PNC was positively associated with an increase in CRP (1.188-fold change per 1000 UFP cm-3 day/day (95% credible interval 1.077 to 1.299)) and an increase in FeNO among atopic participants (1.054 fold change per 1000 UFP cm-3 day/day (95% CrI 1.005 to 1.106)). UFPs do not affect respiratory health outcomes in children but do have systemic effects, detected here in the form of a positive association with a biomarker for systemic inflammation. This is consistent with the known propensity of UFPs to penetrate deep into the lung and circulatory system.