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Artigo em Chinês | WPRIM (Pacífico Ocidental) | ID: wpr-580800

RESUMO

0.05),compared with the model group respectively.At the pathological stage of atypical hyperplasia,the expressing activity of Bcl-2 was significantly lower(P 0.05).There was a parallel tendency of expressing activity in either Bax mRNA or Bcl-2 mRNA with Bax or Bcl-2 itself respectively in any stage of pathology.Conclusion The anticancer effect of QBTRF was realized by regulating mitochondrion pathway in the developing process of carcinogenesis among rats.

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