Impaired intramembranous bone formation during bone repair in the absence of tumor necrosis factor-alpha signaling.

Gerstenfeld, L C; Cho, T J; Kon, T; Aizawa, T; Cruceta, J; Graves, B D; Einhorn, T A

Gerstenfeld, L C; Cho, T J; Kon, T; Aizawa, T; Cruceta, J; Graves, B D; Einhorn, T A.

Affiliation

Gerstenfeld LC; Department of Orthopaedic Surgery, Boston University Medical Center, Boston, Mass., USA. lgersten@bu.edu

Cells Tissues Organs ; 169(3): 285-94, 2001.

Article in En | MEDLINE | ID: mdl-11455125

Subject(s)

Fracture Healing/physiology; Osteogenesis/physiology; Tumor Necrosis Factor-alpha/physiology; Animals; Antigens, CD/genetics; Antigens, CD/physiology; Collagen/genetics; Fracture Healing/genetics; Gene Expression; Glyceraldehyde-3-Phosphate Dehydrogenases/genetics; Male; Mice; Mice, Knockout; Osteocalcin/genetics; Osteogenesis/genetics; RNA, Messenger/genetics; RNA, Messenger/metabolism; Receptors, Tumor Necrosis Factor/genetics; Receptors, Tumor Necrosis Factor/physiology; Receptors, Tumor Necrosis Factor, Type I; Receptors, Tumor Necrosis Factor, Type II; Signal Transduction

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Collection: 01-internacional Database: MEDLINE Main subject: Osteogenesis / Tumor Necrosis Factor-alpha / Fracture Healing Type of study: Prognostic_studies Limits: Animals Language: En Journal: Cells Tissues Organs Journal subject: ANATOMIA Year: 2001 Document type: Article Affiliation country: United States Country of publication: Switzerland

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