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An AT1-receptor antagonist and an angiotensin-converting enzyme inhibitor protect against hypoxia-induced apoptosis in human aortic endothelial cells through upregulation of endothelial cell nitric oxide synthase activity.
Matsumoto, Naoyuki; Manabe, Hiroki; Ochiai, Jun; Fujita, Noriko; Takagi, Tomohisa; Uemura, Manabu; Naito, Yuji; Yoshida, Norimasa; Oka, Shigenori; Yoshikawa, Toshikazu.
Affiliation
  • Matsumoto N; First Department of Internal Medicine, Kyoto Prefectural University of Medicine, Kawaramachi-Hirokoji, Kamigyo-ku, Kyoto, 602-0841, Japan.
Shock ; 19(6): 547-52, 2003 Jun.
Article in En | MEDLINE | ID: mdl-12785010
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Collection: 01-internacional Database: MEDLINE Main subject: Tetrazoles / Thiazepines / Benzimidazoles / Angiotensin-Converting Enzyme Inhibitors / Endothelium, Vascular / Apoptosis / Nitric Oxide Synthase / Angiotensin II Type 1 Receptor Blockers Limits: Humans Language: En Journal: Shock Journal subject: ANGIOLOGIA / CARDIOLOGIA Year: 2003 Document type: Article Affiliation country: Japan Country of publication: United States
Search on Google
Add to My VHL
Print
XML
PubMed Links

Collection: 01-internacional Database: MEDLINE Main subject: Tetrazoles / Thiazepines / Benzimidazoles / Angiotensin-Converting Enzyme Inhibitors / Endothelium, Vascular / Apoptosis / Nitric Oxide Synthase / Angiotensin II Type 1 Receptor Blockers Limits: Humans Language: En Journal: Shock Journal subject: ANGIOLOGIA / CARDIOLOGIA Year: 2003 Document type: Article Affiliation country: Japan Country of publication: United States