Matrix metalloproteases: degradation of the inhibitory environment of the transected optic nerve and the scar by regenerating axons.

Ahmed, Zubair; Dent, Russell G; Leadbeater, Wendy E; Smith, Conrad; Berry, Martin; Logan, Ann

Ahmed, Zubair; Dent, Russell G; Leadbeater, Wendy E; Smith, Conrad; Berry, Martin; Logan, Ann.

Affiliation

Ahmed Z; Molecular Neuroscience Group, Department of Medicine, University of Birmingham, Birmingham B15 2TT, UK. z.ahmed.1@bham.ac.uk

Mol Cell Neurosci ; 28(1): 64-78, 2005 Jan.

Article in En | MEDLINE | ID: mdl-15607942

Subject(s)

Cicatrix/enzymology; Matrix Metalloproteinases/metabolism; Nerve Regeneration/physiology; Optic Nerve Injuries/enzymology; Optic Nerve/enzymology; Animals; Astrocytes/metabolism; Cicatrix/physiopathology; Disease Models, Animal; Down-Regulation/physiology; Female; GAP-43 Protein/metabolism; Gelatinases/metabolism; Growth Cones/metabolism; Optic Nerve/physiopathology; Optic Nerve Injuries/physiopathology; Rats; Rats, Inbred F344; Tissue Inhibitor of Metalloproteinase-1/metabolism; Tissue Inhibitor of Metalloproteinase-2/metabolism; Up-Regulation/physiology

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Collection: 01-internacional Database: MEDLINE Main subject: Optic Nerve / Cicatrix / Optic Nerve Injuries / Matrix Metalloproteinases / Nerve Regeneration Limits: Animals Language: En Journal: Mol Cell Neurosci Journal subject: BIOLOGIA MOLECULAR / NEUROLOGIA Year: 2005 Document type: Article Affiliation country: United kingdom Country of publication: United States

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