Estrogenic compounds inhibit gap junctional intercellular communication in mouse Leydig TM3 cells.

Iwase, Yumiko; Fukata, Hideki; Mori, Chisato

Iwase, Yumiko; Fukata, Hideki; Mori, Chisato.

Affiliation

Iwase Y; Department of Bioenvironmental Medicine, Graduate School of Medicine, Chiba University, Chiba 260-8670, Japan. Iwase.Yumiko@mg.m-pharma.co.jp

Toxicol Appl Pharmacol ; 212(3): 237-46, 2006 May 01.

Article in En | MEDLINE | ID: mdl-16199072

Subject(s)

Diethylstilbestrol/toxicity; Estradiol/toxicity; Estrogens/toxicity; Gap Junctions/drug effects; Genistein/toxicity; Leydig Cells/drug effects; Animals; Cell Line; Dose-Response Relationship, Drug; Drug Combinations; Enzyme Inhibitors/pharmacology; Estradiol/analogs & derivatives; Estradiol/pharmacology; Estrogen Antagonists/pharmacology; Fluorescent Dyes/metabolism; Fulvestrant; Gap Junctions/metabolism; Isoquinolines/metabolism; Leydig Cells/metabolism; Leydig Cells/pathology; Male; Mice; Naphthalenes/pharmacology; Protein Kinase C/antagonists & inhibitors; Protein Kinase C/metabolism; Receptors, Estrogen/antagonists & inhibitors; Receptors, Estrogen/metabolism

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Collection: 01-internacional Database: MEDLINE Main subject: Gap Junctions / Genistein / Diethylstilbestrol / Estradiol / Estrogens / Leydig Cells Limits: Animals Language: En Journal: Toxicol Appl Pharmacol Year: 2006 Document type: Article Affiliation country: Japan Country of publication: United States

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