Lack of UCP2 reduces Fas-mediated liver injury in ob/ob mice and reveals importance of cell-specific UCP2 expression.

Fülöp, Péter; Derdák, Zoltán; Sheets, Anthony; Sabo, Edmond; Berthiaume, Eric P; Resnick, Murray B; Wands, Jack R; Paragh, György; Baffy, György

Fülöp, Péter; Derdák, Zoltán; Sheets, Anthony; Sabo, Edmond; Berthiaume, Eric P; Resnick, Murray B; Wands, Jack R; Paragh, György; Baffy, György.

Affiliation

Fülöp P; Division of Gastroenterology & Liver Research Center, Brown Medical School and Rhode Island Hospital, Providence, Rhode Island 02903, USA.

Hepatology ; 44(3): 592-601, 2006 Sep.

Article in En | MEDLINE | ID: mdl-16941708

Subject(s)

DNA/genetics; Fatty Liver/metabolism; Gene Expression; Membrane Transport Proteins/genetics; Mitochondrial Proteins/genetics; Acute Disease; Animals; Apoptosis; Disease Models, Animal; Fatty Liver/genetics; Fatty Liver/pathology; Female; Hepatocytes/metabolism; Hepatocytes/pathology; Ion Channels; Membrane Transport Proteins/metabolism; Mice; Mitochondrial Proteins/metabolism; Oxidative Stress; Polymerase Chain Reaction; Uncoupling Protein 2; fas Receptor/toxicity

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Collection: 01-internacional Database: MEDLINE Main subject: Membrane Transport Proteins / DNA / Gene Expression / Mitochondrial Proteins / Fatty Liver Type of study: Prognostic_studies Limits: Animals Language: En Journal: Hepatology Year: 2006 Document type: Article Affiliation country: United States Country of publication: United States

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