Protecting against antimicrobial effectors in the phagosome allows SodCII to contribute to virulence in Salmonella enterica serovar Typhimurium.

Kim, Byoungkwan; Richards, Susan M; Gunn, John S; Slauch, James M

Kim, Byoungkwan; Richards, Susan M; Gunn, John S; Slauch, James M.

Affiliation

Kim B; Department of Microbiology, University of Illinois, B103 Chemical and Life Sciences Laboratory, 601 S. Goodwin Ave., Urbana, IL 61801, USA.

J Bacteriol ; 192(8): 2140-9, 2010 Apr.

Article in En | MEDLINE | ID: mdl-20154132

Subject(s)

Bacterial Proteins/metabolism; Phagosomes/microbiology; Salmonella typhimurium/metabolism; Salmonella typhimurium/pathogenicity; Superoxide Dismutase/physiology; Virulence/physiology; Animals; Bacterial Proteins/genetics; Blotting, Western; Cell Line; Female; Macrophages/microbiology; Mice; Mice, Inbred BALB C; Osmotic Pressure/physiology; Periplasm/enzymology; Polymyxin B/pharmacology; Salmonella typhimurium/drug effects; Spleen/microbiology; Superoxide Dismutase/genetics; Virulence/genetics

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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Salmonella typhimurium / Superoxide Dismutase / Bacterial Proteins / Virulence / Phagosomes Limits: Animals Language: En Journal: J Bacteriol Year: 2010 Document type: Article Affiliation country: United States Country of publication: United States

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