Long-term electromagnetic field treatment enhances brain mitochondrial function of both Alzheimer's transgenic mice and normal mice: a mechanism for electromagnetic field-induced cognitive benefit?

Dragicevic, N; Bradshaw, P C; Mamcarz, M; Lin, X; Wang, L; Cao, C; Arendash, G W

Dragicevic, N; Bradshaw, P C; Mamcarz, M; Lin, X; Wang, L; Cao, C; Arendash, G W.

Affiliation

Dragicevic N; Department of Cell Biology, University of South Florida, FL 33620, USA.

Neuroscience ; 185: 135-49, 2011 Jun 30.

Article in En | MEDLINE | ID: mdl-21514369

Subject(s)

Alzheimer Disease/pathology; Brain/ultrastructure; Magnetic Field Therapy/methods; Mitochondria/radiation effects; Adenosine Triphosphate/metabolism; Alzheimer Disease/genetics; Amyloid beta-Peptides/metabolism; Amyloid beta-Protein Precursor/genetics; Analysis of Variance; Animals; Body Temperature/genetics; Brain/pathology; Brain/radiation effects; Disease Models, Animal; Electron Transport Complex IV/metabolism; Enzyme-Linked Immunosorbent Assay/methods; Humans; Membrane Potential, Mitochondrial/genetics; Mice; Mice, Transgenic; Mutation/genetics; Peptide Fragments/metabolism; Presenilin-1/genetics; Reactive Oxygen Species/metabolism

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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Brain / Magnetic Field Therapy / Alzheimer Disease / Mitochondria Type of study: Prognostic_studies Limits: Animals / Humans Language: En Journal: Neuroscience Year: 2011 Document type: Article Affiliation country: United States Country of publication: United States

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