Parvovirus B19 infection of human primary erythroid progenitor cells triggers ATR-Chk1 signaling, which promotes B19 virus replication.

Luo, Yong; Lou, Sai; Deng, Xuefeng; Liu, Zhengwen; Li, Yi; Kleiboeker, Steve; Qiu, Jianming

Luo, Yong; Lou, Sai; Deng, Xuefeng; Liu, Zhengwen; Li, Yi; Kleiboeker, Steve; Qiu, Jianming.

Affiliation

Luo Y; Department of Microbiology, Molecular Genetics and Immunology, University of Kansas Medical Center, Kansas City, KS 66160, USA. jqiu@kumc.edu

J Virol ; 85(16): 8046-55, 2011 Aug.

Article in En | MEDLINE | ID: mdl-21680529

Subject(s)

Cell Cycle Proteins/metabolism; Erythroid Precursor Cells/virology; Parvovirus B19, Human/physiology; Protein Kinases/metabolism; Protein Serine-Threonine Kinases/metabolism; Virus Replication; Ataxia Telangiectasia Mutated Proteins; Calcium-Binding Proteins/metabolism; Cell Cycle; Cell Line; Checkpoint Kinase 1; DNA Damage; DNA Repair; DNA-Binding Proteins/metabolism; Flow Cytometry; Fluorescent Antibody Technique; Histones/metabolism; Humans; In Situ Hybridization, Fluorescence; Parvovirus B19, Human/genetics; Parvovirus B19, Human/metabolism; Phosphorylation; Replication Protein A/metabolism; Signal Transduction; Tumor Suppressor Proteins/metabolism

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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Protein Kinases / Virus Replication / Erythroid Precursor Cells / Parvovirus B19, Human / Protein Serine-Threonine Kinases / Cell Cycle Proteins Limits: Humans Language: En Journal: J Virol Year: 2011 Document type: Article Affiliation country: United States Country of publication: United States

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