Entry of herpes simplex virus type 1 (HSV-1) into the distal axons of trigeminal neurons favors the onset of nonproductive, silent infection.

Hafezi, Wali; Lorentzen, Eva U; Eing, Bodo R; Müller, Marcus; King, Nicholas J C; Klupp, Barbara; Mettenleiter, Thomas C; Kühn, Joachim E

Hafezi, Wali; Lorentzen, Eva U; Eing, Bodo R; Müller, Marcus; King, Nicholas J C; Klupp, Barbara; Mettenleiter, Thomas C; Kühn, Joachim E.

Affiliation

Hafezi W; University Hospital Münster, Institute of Medical Microbiology-Clinical Virology, Münster, Germany.

PLoS Pathog ; 8(5): e1002679, 2012.

Article in En | MEDLINE | ID: mdl-22589716

Subject(s)

Axons/virology; Herpesvirus 1, Human/pathogenicity; Trigeminal Ganglion/virology; Virus Internalization; Virus Latency; Acetamides/pharmacology; Animals; Asymptomatic Infections; Cells, Cultured; Chick Embryo; Gene Expression Regulation, Viral; Herpes Simplex Virus Protein Vmw65/metabolism; Herpesvirus 1, Suid/pathogenicity; Herpesvirus 2, Human/pathogenicity; Immediate-Early Proteins/genetics; Transcriptional Activation; Virus Replication

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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Axons / Trigeminal Ganglion / Virus Latency / Herpesvirus 1, Human / Virus Internalization Type of study: Prognostic_studies Limits: Animals Language: En Journal: PLoS Pathog Year: 2012 Document type: Article Affiliation country: Germany Country of publication: United States

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