Helicobacter pylori-induced alteration of epithelial cell signaling and polarity: a possible mechanism of gastric carcinoma etiology and disparity.

Osman, Mahasin A; Bloom, George S; Tagoe, Emmanuel A

Osman, Mahasin A; Bloom, George S; Tagoe, Emmanuel A.

Affiliation

Osman MA; Department of Molecular Pharmacology, Physiology and Biotechnology, Warren Alpert Medical School, Brown University, Providence, RI 02912, USA. mahasin_osman@Brown.edu

Cytoskeleton (Hoboken) ; 70(7): 349-59, 2013 Jul.

Article in En | MEDLINE | ID: mdl-23629919

Subject(s)

Cell Communication/physiology; Helicobacter Infections/microbiology; Helicobacter Infections/pathology; Helicobacter pylori/physiology; Stomach Neoplasms/microbiology; Stomach Neoplasms/pathology; Animals; Cell Polarity/physiology; Epithelial Cells/microbiology; Epithelial Cells/pathology; Humans

Key words

Akt; Cdc42; Erk1/2; H. pylori; IQGAP; gastric cancer; mTOR

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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Stomach Neoplasms / Cell Communication / Helicobacter pylori / Helicobacter Infections Type of study: Etiology_studies Limits: Animals / Humans Language: En Journal: Cytoskeleton (Hoboken) Year: 2013 Document type: Article Affiliation country: United States

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