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Protein kinase B (PKB/AKT1) formed signaling complexes with mitochondrial proteins and prevented glycolytic energy dysfunction in cultured cardiomyocytes during ischemia-reperfusion injury.
Deng, Wu; Leu, Hsin-Bang; Chen, Yumay; Chen, Yu-Han; Epperson, Christine M; Juang, Charity; Wang, Ping H.
Affiliation
  • Deng W; University of California Irvine Diabetes Center and Department of Medicine (W.D., H.-B.L., Y.C., Y.-H.C., C.M.E., C.J., P.H.W.), University of California Irvine, California 92697; Department of Medicine (H.-B.L.), Veterans General Hospital, and School of Medicine, National Yang-Ming University, Taipei, 11217, Taiwan; and Departments of Biological Chemistry and Physiology and Biophysics (P.H.W.), University of California Irvine, California 92697.
Endocrinology ; 155(5): 1618-28, 2014 May.
Article in En | MEDLINE | ID: mdl-24601882
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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Pyruvate Dehydrogenase Complex / Myocardial Reperfusion Injury / Signal Transduction / Myocytes, Cardiac / Proto-Oncogene Proteins c-akt / Glycolysis Type of study: Prognostic_studies Limits: Animals Language: En Journal: Endocrinology Year: 2014 Document type: Article Country of publication: United States
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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Pyruvate Dehydrogenase Complex / Myocardial Reperfusion Injury / Signal Transduction / Myocytes, Cardiac / Proto-Oncogene Proteins c-akt / Glycolysis Type of study: Prognostic_studies Limits: Animals Language: En Journal: Endocrinology Year: 2014 Document type: Article Country of publication: United States