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Astroglial glutamate transporter deficiency increases synaptic excitability and leads to pathological repetitive behaviors in mice.
Aida, Tomomi; Yoshida, Junichi; Nomura, Masatoshi; Tanimura, Asami; Iino, Yusuke; Soma, Miho; Bai, Ning; Ito, Yukiko; Cui, Wanpeng; Aizawa, Hidenori; Yanagisawa, Michiko; Nagai, Terumi; Takata, Norio; Tanaka, Kenji F; Takayanagi, Ryoichi; Kano, Masanobu; Götz, Magdalena; Hirase, Hajime; Tanaka, Kohichi.
Affiliation
  • Aida T; Laboratory of Molecular Neuroscience, Medical Research Institute, Tokyo Medical and Dental University, Tokyo, Japan.
  • Yoshida J; Laboratory of Molecular Neuroscience, Medical Research Institute, Tokyo Medical and Dental University, Tokyo, Japan.
  • Nomura M; Department of Medicine and Bioregulatory Science, Graduate School of Medical Sciences, Kyushu University, Fukuoka, Japan.
  • Tanimura A; Department of Neurophysiology, Graduate School of Medicine, University of Tokyo, Tokyo, Japan.
  • Iino Y; Laboratory of Molecular Neuroscience, Medical Research Institute, Tokyo Medical and Dental University, Tokyo, Japan.
  • Soma M; Laboratory of Molecular Neuroscience, Medical Research Institute, Tokyo Medical and Dental University, Tokyo, Japan.
  • Bai N; Laboratory of Molecular Neuroscience, Medical Research Institute, Tokyo Medical and Dental University, Tokyo, Japan.
  • Ito Y; Laboratory of Molecular Neuroscience, Medical Research Institute, Tokyo Medical and Dental University, Tokyo, Japan.
  • Cui W; Laboratory of Molecular Neuroscience, Medical Research Institute, Tokyo Medical and Dental University, Tokyo, Japan.
  • Aizawa H; Laboratory of Molecular Neuroscience, Medical Research Institute, Tokyo Medical and Dental University, Tokyo, Japan.
  • Yanagisawa M; Laboratory of Molecular Neuroscience, Medical Research Institute, Tokyo Medical and Dental University, Tokyo, Japan.
  • Nagai T; Laboratory for Neuron-Glia Circuitry, Brain Science Institute, RIKEN, Saitama, Japan.
  • Takata N; Laboratory for Neuron-Glia Circuitry, Brain Science Institute, RIKEN, Saitama, Japan.
  • Tanaka KF; Department of Neuropsychiatry, Keio University School of Medicine, Tokyo, Japan.
  • Takayanagi R; Department of Medicine and Bioregulatory Science, Graduate School of Medical Sciences, Kyushu University, Fukuoka, Japan.
  • Kano M; Department of Neurophysiology, Graduate School of Medicine, University of Tokyo, Tokyo, Japan.
  • Götz M; Physiological Genomics, Institute of Physiology, Ludwig-Maximilians University Munich, Munich, Germany.
  • Hirase H; Department of Neuropsychiatry, Keio University School of Medicine, Tokyo, Japan.
  • Tanaka K; 1] Laboratory of Molecular Neuroscience, Medical Research Institute, Tokyo Medical and Dental University, Tokyo, Japan [2] JST, CREST, Saitama, Japan [3] The Center for Brain Integration Research, Tokyo Medical and Dental University, Tokyo, Japan.
Neuropsychopharmacology ; 40(7): 1569-79, 2015 Jun.
Article in En | MEDLINE | ID: mdl-25662838
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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Synapses / Cumulative Trauma Disorders / Cerebral Cortex / Excitatory Amino Acid Transporter 1 / Excitatory Amino Acid Transporter 2 Type of study: Etiology_studies / Prognostic_studies Limits: Animals Language: En Journal: Neuropsychopharmacology Journal subject: NEUROLOGIA / PSICOFARMACOLOGIA Year: 2015 Document type: Article Affiliation country: Japan Country of publication: United kingdom
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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Synapses / Cumulative Trauma Disorders / Cerebral Cortex / Excitatory Amino Acid Transporter 1 / Excitatory Amino Acid Transporter 2 Type of study: Etiology_studies / Prognostic_studies Limits: Animals Language: En Journal: Neuropsychopharmacology Journal subject: NEUROLOGIA / PSICOFARMACOLOGIA Year: 2015 Document type: Article Affiliation country: Japan Country of publication: United kingdom