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ERK reinforces actin polymerization to power persistent edge protrusion during motility.
Mendoza, Michelle C; Vilela, Marco; Juarez, Jesus E; Blenis, John; Danuser, Gaudenz.
Affiliation
  • Mendoza MC; Department of Cell Biology, Harvard Medical School, Boston, MA 02115, USA. michelle.mendoza@ucsf.edu gaudenz.danuser@utsouthwestern.edu.
  • Vilela M; Department of Cell Biology, Harvard Medical School, Boston, MA 02115, USA.
  • Juarez JE; Departments of Cell and Tissue Biology and Pathology, University of California, San Francisco, 513 Parnassus Avenue, San Francisco, CA 94143, USA.
  • Blenis J; Department of Cell Biology, Harvard Medical School, Boston, MA 02115, USA.
  • Danuser G; Department of Cell Biology, Harvard Medical School, Boston, MA 02115, USA. michelle.mendoza@ucsf.edu gaudenz.danuser@utsouthwestern.edu.
Sci Signal ; 8(377): ra47, 2015 May 19.
Article in En | MEDLINE | ID: mdl-25990957
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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Cell Movement / Actins / MAP Kinase Signaling System / Cell Surface Extensions / Actin-Related Protein 2-3 Complex / Models, Biological Type of study: Prognostic_studies Limits: Humans Language: En Journal: Sci Signal Journal subject: CIENCIA / FISIOLOGIA Year: 2015 Document type: Article Country of publication: United States
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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Cell Movement / Actins / MAP Kinase Signaling System / Cell Surface Extensions / Actin-Related Protein 2-3 Complex / Models, Biological Type of study: Prognostic_studies Limits: Humans Language: En Journal: Sci Signal Journal subject: CIENCIA / FISIOLOGIA Year: 2015 Document type: Article Country of publication: United States