Behavioral defects in C. elegans egl-36 mutants result from potassium channels shifted in voltage-dependence of activation. | Neuron;19(1): 151-64, 1997 Jul. | MEDLINE

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Behavioral defects in C. elegans egl-36 mutants result from potassium channels shifted in voltage-dependence of activation.

Johnstone, D B; Wei, A; Butler, A; Salkoff, L; Thomas, J H.

Affiliation

Johnstone DB; Department of Genetics, University of Washington, Seattle 98195, USA.

Neuron ; 19(1): 151-64, 1997 Jul.

Article in En | MEDLINE | ID: mdl-9247271

Subject(s)

Genes/genetics; Membrane Potentials/physiology; Mutation/genetics; Potassium Channels/genetics; Animals; Base Sequence; Molecular Sequence Data; Oocytes; Patch-Clamp Techniques; Phenotype; Xenopus

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Collection: 01-internacional Database: MEDLINE Main subject: Potassium Channels / Genes / Membrane Potentials / Mutation Limits: Animals Language: En Journal: Neuron Journal subject: NEUROLOGIA Year: 1997 Document type: Article Affiliation country: United States

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Collection: 01-internacional Database: MEDLINE Main subject: Potassium Channels / Genes / Membrane Potentials / Mutation Limits: Animals Language: En Journal: Neuron Journal subject: NEUROLOGIA Year: 1997 Document type: Article Affiliation country: United States