Licorice-nduced Hypokalemia and Myopathy / 대한신장학회잡지
Korean Journal of Nephrology
; : 651-655, 2006.
Article
in Korean
| WPRIM (Western Pacific)
| ID: wpr-176122
Responsible library:
WPRO
ABSTRACT
Chronic ingestion of licorice or licorice-like compounds induces a syndrome with typical findings of mineralocorticoid excess such as hypertension, hypokalemia, metabolic alkalosis, low plasma renin activity. The only unique feature is that plasma aldosterone concentration is decreased. We described a 79-year-old woman who, with a plasma K+ 1.75 mEq/L, showed a paralysis and severe rhabdomyolysis after the habitual comsumption of licorice in the form of a herbal medication. Following potassium replacement therapy and discontinuation of licorice ingestion, complete clinical recovery was observed within ten days. It is important for physicians to keep licorice consumption in mind as a cause for hypokalemic paralysis and rhabdomyolysis.
Full text:
Available
Database:
WPRIM (Western Pacific)
Main subject:
Paralysis
/
Plasma
/
Potassium
/
Rhabdomyolysis
/
Renin
/
Mineralocorticoid Excess Syndrome, Apparent
/
Eating
/
Aldosterone
/
Alkalosis
/
Glycyrrhiza
Limits:
Aged
/
Female
/
Humans
Language:
Korean
Journal:
Korean Journal of Nephrology
Year:
2006
Document type:
Article