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Inhibition of bleomycin-induced pulmonary fibrosis by bone marrow-derived mesenchymal stem cells might be mediated by decreasing MMP9, TIMP-1, INF-γ and TGF-ß.
Yu, Shi-huan; Liu, Li-jie; Lv, Bin; Che, Chun-li; Fan, Da-ping; Wang, Li-feng; Zhang, Yi-mei.
Afiliación
  • Yu SH; Department of Pulmonary Disease, The First Affiliated Hospital of Harbin Medical University, Harbin, China.
  • Liu LJ; Department of Pulmonary Disease, The Fourth Affiliated Hospital of Harbin Medical University, Harbin, China.
  • Lv B; Department of Pulmonary Disease, The First Affiliated Hospital of Harbin Medical University, Harbin, China.
  • Che CL; Department of Pulmonary Disease, The First Affiliated Hospital of Harbin Medical University, Harbin, China.
  • Fan DP; Department of Pulmonary Disease, The First Affiliated Hospital of Harbin Medical University, Harbin, China.
  • Wang LF; Department of Pathology, The First Affiliated Hospital of Harbin Medical University, Harbin, China.
  • Zhang YM; Department of Pulmonary Disease, The First Affiliated Hospital of Harbin Medical University, Harbin, China.
Cell Biochem Funct ; 33(6): 356-66, 2015 Aug.
Article en En | MEDLINE | ID: mdl-26178702
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Fibrosis Pulmonar / Interferón gamma / Inhibidor Tisular de Metaloproteinasa-1 / Metaloproteinasa 9 de la Matriz / Factor de Crecimiento Transformador beta1 / Células Madre Mesenquimatosas Tipo de estudio: Prognostic_studies Límite: Animals Idioma: En Revista: Cell Biochem Funct Año: 2015 Tipo del documento: Article País de afiliación: China Pais de publicación: Reino Unido
Texto completo
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Fibrosis Pulmonar / Interferón gamma / Inhibidor Tisular de Metaloproteinasa-1 / Metaloproteinasa 9 de la Matriz / Factor de Crecimiento Transformador beta1 / Células Madre Mesenquimatosas Tipo de estudio: Prognostic_studies Límite: Animals Idioma: En Revista: Cell Biochem Funct Año: 2015 Tipo del documento: Article País de afiliación: China Pais de publicación: Reino Unido