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Novel mechanisms of PIEZO1 dysfunction in hereditary xerocytosis.
Glogowska, Edyta; Schneider, Eve R; Maksimova, Yelena; Schulz, Vincent P; Lezon-Geyda, Kimberly; Wu, John; Radhakrishnan, Kottayam; Keel, Siobán B; Mahoney, Donald; Freidmann, Alison M; Altura, Rachel A; Gracheva, Elena O; Bagriantsev, Sviatoslav N; Kalfa, Theodosia A; Gallagher, Patrick G.
Afiliación
  • Glogowska E; Department of Pediatrics and.
  • Schneider ER; Department of Cellular and Molecular Physiology, Yale University School of Medicine, New Haven, CT.
  • Maksimova Y; Department of Pediatrics and.
  • Schulz VP; Department of Pediatrics and.
  • Lezon-Geyda K; Department of Pediatrics and.
  • Wu J; Division of Hematology, Oncology and Bone Marrow Transplant, Department of Pediatrics, University of British Columbia, Vancouver, BC, Canada.
  • Radhakrishnan K; Department of Paediatric Haematology/Oncology, Children's Cancer Centre, Monash Children's Hospital, Melbourne, VIC, Australia.
  • Keel SB; Division of Hematology, Department of Medicine, University of Washington, Seattle, WA.
  • Mahoney D; Section of Hematology/Oncology, Department of Pediatrics, Baylor College of Medicine, Texas Children's Hospital, Houston, TX.
  • Freidmann AM; Division of Pediatric Hematology and Oncology, Department of Pediatrics, Massachusetts General Hospital, Boston, MA.
  • Altura RA; Division of Pediatric Hematology-Oncology, Department of Pediatrics, Warren Albert School of Medicine, Brown University, Providence, RI.
  • Gracheva EO; Department of Cellular and Molecular Physiology, Yale University School of Medicine, New Haven, CT.
  • Bagriantsev SN; Department of Neuroscience and.
  • Kalfa TA; Program in Cellular Neuroscience, Neurodegeneration and Repair, Yale University, New Haven, CT.
  • Gallagher PG; Department of Cellular and Molecular Physiology, Yale University School of Medicine, New Haven, CT.
Blood ; 130(16): 1845-1856, 2017 10 19.
Article en En | MEDLINE | ID: mdl-28716860
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Hidropesía Fetal / Anemia Hemolítica Congénita / Canales Iónicos Tipo de estudio: Etiology_studies / Incidence_studies / Observational_studies / Prognostic_studies / Risk_factors_studies Límite: Adult / Child / Female / Humans / Male / Newborn Idioma: En Revista: Blood Año: 2017 Tipo del documento: Article Pais de publicación: Estados Unidos
Texto completo
Añadir a Mi BVS
Imprimir
XML
PubMed Links
Buscar en Google

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Hidropesía Fetal / Anemia Hemolítica Congénita / Canales Iónicos Tipo de estudio: Etiology_studies / Incidence_studies / Observational_studies / Prognostic_studies / Risk_factors_studies Límite: Adult / Child / Female / Humans / Male / Newborn Idioma: En Revista: Blood Año: 2017 Tipo del documento: Article Pais de publicación: Estados Unidos