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Blockade of CTLA-4 and Tim-3 pathways induces fetal loss with altered cytokine profiles by decidual CD4+T cells.
Wang, Songcun; Chen, Chunqin; Li, Mengdie; Qian, Jinfeng; Sun, Fengyun; Li, Yunyun; Yu, Min; Wang, Mingyan; Zang, Xingxing; Zhu, Rui; Li, Dajin; Du, Meirong.
Afiliación
  • Wang S; Laboratory for Reproductive Immunology, NHC Key Lab of Reproduction Regulation(Shanghai Institute of Planned Parenthood Research), Shanghai Key Laboratory of Female Reproductive Endocrine Related Diseases, Hospital of Obstetrics and Gynecology, Fudan University Shanghai Medical College, Shanghai, P.
  • Chen C; Laboratory for Reproductive Immunology, NHC Key Lab of Reproduction Regulation(Shanghai Institute of Planned Parenthood Research), Shanghai Key Laboratory of Female Reproductive Endocrine Related Diseases, Hospital of Obstetrics and Gynecology, Fudan University Shanghai Medical College, Shanghai, P.
  • Li M; Laboratory for Reproductive Immunology, NHC Key Lab of Reproduction Regulation(Shanghai Institute of Planned Parenthood Research), Shanghai Key Laboratory of Female Reproductive Endocrine Related Diseases, Hospital of Obstetrics and Gynecology, Fudan University Shanghai Medical College, Shanghai, P.
  • Qian J; Laboratory for Reproductive Immunology, NHC Key Lab of Reproduction Regulation(Shanghai Institute of Planned Parenthood Research), Shanghai Key Laboratory of Female Reproductive Endocrine Related Diseases, Hospital of Obstetrics and Gynecology, Fudan University Shanghai Medical College, Shanghai, P.
  • Sun F; Laboratory for Reproductive Immunology, NHC Key Lab of Reproduction Regulation(Shanghai Institute of Planned Parenthood Research), Shanghai Key Laboratory of Female Reproductive Endocrine Related Diseases, Hospital of Obstetrics and Gynecology, Fudan University Shanghai Medical College, Shanghai, P.
  • Li Y; Laboratory for Reproductive Immunology, NHC Key Lab of Reproduction Regulation(Shanghai Institute of Planned Parenthood Research), Shanghai Key Laboratory of Female Reproductive Endocrine Related Diseases, Hospital of Obstetrics and Gynecology, Fudan University Shanghai Medical College, Shanghai, P.
  • Yu M; Reproductive Medicine Center, Hospital of Obstetrics and Gynecology, FudanUniversity Shanghai Medical School, Shanghai, P.R. China.
  • Wang M; Department of Clinical Laboratory, Hospital of Obstetrics and Gynecology, Fudan University, Shanghai, P.R. China.
  • Zang X; Department of Medicine, Montefiore Medical Center, Albert Einstein College of Medicine, Bronx, NY, USA.
  • Zhu R; The Affiliated Suzhou Hospital of Nanjing Medical University/Suzhou Municipal Hospital, Suzhou, 215008, China.
  • Li D; Laboratory for Reproductive Immunology, NHC Key Lab of Reproduction Regulation(Shanghai Institute of Planned Parenthood Research), Shanghai Key Laboratory of Female Reproductive Endocrine Related Diseases, Hospital of Obstetrics and Gynecology, Fudan University Shanghai Medical College, Shanghai, P.
  • Du M; Laboratory for Reproductive Immunology, NHC Key Lab of Reproduction Regulation(Shanghai Institute of Planned Parenthood Research), Shanghai Key Laboratory of Female Reproductive Endocrine Related Diseases, Hospital of Obstetrics and Gynecology, Fudan University Shanghai Medical College, Shanghai, P.
Cell Death Dis ; 10(1): 15, 2019 01 08.
Article en En | MEDLINE | ID: mdl-30622243
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Linfocitos T CD4-Positivos / Aborto Espontáneo / Citocinas / Decidua / Antígeno CTLA-4 / Receptor 2 Celular del Virus de la Hepatitis A Límite: Animals / Female / Humans / Male / Pregnancy Idioma: En Revista: Cell Death Dis Año: 2019 Tipo del documento: Article Pais de publicación: Reino Unido
Texto completo
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XML
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Linfocitos T CD4-Positivos / Aborto Espontáneo / Citocinas / Decidua / Antígeno CTLA-4 / Receptor 2 Celular del Virus de la Hepatitis A Límite: Animals / Female / Humans / Male / Pregnancy Idioma: En Revista: Cell Death Dis Año: 2019 Tipo del documento: Article Pais de publicación: Reino Unido