Sialomucin CD43 Plays a Deleterious Role in the Development of Experimental Heart Failure Induced by Pressure Overload by Modulating Cardiac Inflammation and Fibrosis.

Kaur, Kuljeet; Velázquez, Francisco E; Anastasiou, Marina; Ngwenyama, Njabulo; Smolgovsky, Sasha; Aronovitz, Mark; Alcaide, Pilar

Kaur, Kuljeet; Velázquez, Francisco E; Anastasiou, Marina; Ngwenyama, Njabulo; Smolgovsky, Sasha; Aronovitz, Mark; Alcaide, Pilar.

Afiliación

Kaur K; The Department of Immunology, Tufts University School of Medicine, Boston, MA, United States.
Velázquez FE; The Department of Immunology, Tufts University School of Medicine, Boston, MA, United States.
Anastasiou M; The Department of Immunology, Tufts University School of Medicine, Boston, MA, United States.
Ngwenyama N; Department of Internal Medicine, University of Crete Medical School, Crete, Greece.
Smolgovsky S; The Department of Immunology, Tufts University School of Medicine, Boston, MA, United States.
Aronovitz M; The Department of Immunology, Tufts University School of Medicine, Boston, MA, United States.
Alcaide P; The Department of Immunology, Tufts University School of Medicine, Boston, MA, United States.

Front Physiol ; 12: 780854, 2021.

Article en En | MEDLINE | ID: mdl-34925069

Palabras clave

CD43; CXCL10; T cell; heart failure; inflammation

Texto completo

Añadir a Mi BVS

Imprimir

XML

PubMed Links

Buscar en Google

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Idioma: En Revista: Front Physiol Año: 2021 Tipo del documento: Article País de afiliación: Estados Unidos Pais de publicación: Suiza

Texto completo

Añadir a Mi BVS

Imprimir

XML

PubMed Links

Buscar en Google