Docking and stability defects in mitofusin highlight the proteasome as a potential therapeutic target.

Buntenbroich, Ira; Anton, Vincent; Perez-Hernandez, Daniel; Simões, Tânia; Gaedke, Felix; Schauss, Astrid; Dittmar, Gunnar; Riemer, Jan; Escobar-Henriques, Mafalda

Buntenbroich, Ira; Anton, Vincent; Perez-Hernandez, Daniel; Simões, Tânia; Gaedke, Felix; Schauss, Astrid; Dittmar, Gunnar; Riemer, Jan; Escobar-Henriques, Mafalda.

Afiliación

Buntenbroich I; Institute for Genetics,University of Cologne, Cologne 50931, Germany.
Anton V; Institute for Genetics,University of Cologne, Cologne 50931, Germany.
Perez-Hernandez D; Proteomics of Cellular Signaling, Luxembourg Institute of Health, Strassen 1445, Luxembourg.
Simões T; Institute for Genetics,University of Cologne, Cologne 50931, Germany.
Gaedke F; Cologne Excellence Cluster on Cellular Stress Responses in Aging-Associated Diseases (CECAD), University of Cologne, Cologne 50931, Germany.
Schauss A; Cologne Excellence Cluster on Cellular Stress Responses in Aging-Associated Diseases (CECAD), University of Cologne, Cologne 50931, Germany.
Dittmar G; Proteomics of Cellular Signaling, Luxembourg Institute of Health, Strassen 1445, Luxembourg.
Riemer J; Cologne Excellence Cluster on Cellular Stress Responses in Aging-Associated Diseases (CECAD), University of Cologne, Cologne 50931, Germany.
Escobar-Henriques M; Institute for Biochemistry, University of Cologne, Cologne 50931, Germany.

iScience ; 26(7): 107014, 2023 Jul 21.

Article en En | MEDLINE | ID: mdl-37416455

Palabras clave

Biological sciences; Cell biology; Genetics; Molecular biology

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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Idioma: En Revista: IScience Año: 2023 Tipo del documento: Article País de afiliación: Alemania Pais de publicación: Estados Unidos

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