Cldn-7 deficiency promotes experimental colitis and associated carcinogenesis by regulating intestinal epithelial integrity.

Wang, Kun; Ding, Yuhan; Xu, Chang; Hao, Mengdi; Li, Huimin; Ding, Lei

Wang, Kun; Ding, Yuhan; Xu, Chang; Hao, Mengdi; Li, Huimin; Ding, Lei.

Afiliação

Wang K; Department of Oncology Surgery, Beijing Shijitan Hospital, Capital Medical University, Beijing, China.
Ding Y; Department of Oncology Surgery, Ninth School of Clinical Medicine, Peking University, Beijing, China.
Xu C; Department of Oncology Surgery, Beijing Shijitan Hospital, Capital Medical University, Beijing, China.
Hao M; Department of Oncology Surgery, Ninth School of Clinical Medicine, Peking University, Beijing, China.
Li H; Department of Oncology Surgery, Beijing Shijitan Hospital, Capital Medical University, Beijing, China.
Ding L; Department of Hepato-Pancreato-Biliary Surgery, Key Laboratory of Carcinogenesis and Translational Research (Ministry of Education/Beijing), Peking University Cancer Hospital and Institute, Beijing, China.

Oncoimmunology ; 10(1): 1923910, 2021 05 11.

Article em En | MEDLINE | ID: mdl-34026335

Assuntos

Colite; Animais; Transformação Celular Neoplásica/genética; Colite/induzido quimicamente; Sulfato de Dextrana/toxicidade; Mucosa Intestinal; Camundongos; Camundongos Endogâmicos C57BL

Palavras-chave

Cldn-7; colitis-associated cancer; inflammation; intestinal epithelial homeostasis

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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Colite Tipo de estudo: Prognostic_studies / Risk_factors_studies Limite: Animals Idioma: En Revista: Oncoimmunology Ano de publicação: 2021 Tipo de documento: Article País de afiliação: China País de publicação: Estados Unidos

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