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Mem. Inst. Oswaldo Cruz ; 100(supl.1): 25-27, Mar. 2005. ilus, tab
Article Dans Anglais | LILACS | ID: lil-402172

Résumé

We have previously reported that in comparison with normal rats, the presence of experimental allergic encephalomyelitis (EAE) leads to decreased endogenous inhibitory activity (EIA) of Ca2+-dependent nitric oxide synthase (NOS) in both brain and serum, and increased expression of protein 3-nitrotyrosine (NT) in brain. In this work we show that animals recovered from the clinical signs of EAE are not different from controls in terms of either brain NOS activity, EIA of NOS, or NT expression. These results suggest that parallel to the reversal of the disease symptoms, a normalization of the production of nitric oxide and related species occurs.


Sujets)
Animaux , Mâle , Rats , Encéphale/enzymologie , Encéphalomyélite auto-immune expérimentale/enzymologie , Nitric oxide synthase type III/métabolisme , Monoxyde d'azote/métabolisme , Dynéines/métabolisme , Encéphalomyélite auto-immune expérimentale/sang , Nitric oxide synthase type III/antagonistes et inhibiteurs , Rats de lignée LEW
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